ATP Synthase c-Subunit Leak Causes Aberrant Cellular Metabolism in Fragile X Syndrome-Reference-Cited by-同舟云学术

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ATP Synthase c-Subunit Leak Causes Aberrant Cellular Metabolism in Fragile X Syndrome

Published:2020-09 Issue:5 Volume:182 Page:1170-1185.e9
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Licznerski Pawel,Park Han-A,Rolyan Harshvardhan,Chen Rongmin,Mnatsakanyan Nelli,Miranda Paige,Graham Morven,Wu Jing,Cruz-Reyes Nicole,Mehta Nikita,Sohail Sana,Salcedo Jorge,Song Erin,Effman Charles,Effman Samuel,Brandao Lucas,Xu Gulan N.,Braker Amber,Gribkoff Valentin K.,Levy Richard J.,Jonas Elizabeth A.

Funder

FRAXA Research Foundation

National Science Foundation

George Cedric Metcalf Charitable Foundation

National Institutes of Health

Simons Foundation Autism Research Initiative

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference104 articles.

1. Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase;Alavian;Nat. Cell Biol.,2011

2. An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore;Alavian;Proc. Natl. Acad. Sci. USA,2014

3. The mitochondrial complex V-associated large-conductance inner membrane current is regulated by cyclosporine and dexpramipexole;Alavian;Mol. Pharmacol.,2015

4. Marble burying and nestlet shredding as tests of repetitive, compulsive-like behaviors in mice;Angoa-Pérez;J. Vis. Exp.,2013

5. Brain-derived neurotrophic factor and neuropsychiatric disorders;Autry;Pharmacol. Rev.,2012

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2. Activation of the 5-HT1A Receptor by Eltoprazine Restores Mitochondrial and Motor Deficits in a Drosophila Model of Fragile X Syndrome;International Journal of Molecular Sciences;2024-08-13

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