Excess Translation of Epigenetic Regulators Contributes to Fragile X Syndrome and Is Alleviated by Brd4 Inhibition-Reference-Cited by-同舟云学术

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Excess Translation of Epigenetic Regulators Contributes to Fragile X Syndrome and Is Alleviated by Brd4 Inhibition

Published:2017-09 Issue:6 Volume:170 Page:1209-1223.e20
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Korb Erica,Herre Margaret,Zucker-Scharff Ilana,Gresack Jodi,Allis C. David,Darnell Robert B.

Funder

Rockefeller University

Robertson Foundation

NIH

Simons Foundation Research Award

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference44 articles.

1. A chromatin-dependent role of the fragile X mental retardation protein FMRP in the DNA damage response;Alpatov;Cell,2014

2. HTSeq--a Python framework to work with high-throughput sequencing data;Anders;Bioinformatics,2015

3. Mutations causing syndromic autism define an axis of synaptic pathophysiology;Auerbach;Nature,2011

4. Double-bromo and extraterminal (BET) domain proteins regulate dendrite morphology and mechanosensory function;Bagley;Genes Dev.,2014

5. Fragile X syndrome: loss of local mRNA regulation alters synaptic development and function;Bassell;Neuron,2008

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