Transcription-Replication Conflict Orientation Modulates R-Loop Levels and Activates Distinct DNA Damage Responses-Reference-Cited by-同舟云学术

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Transcription-Replication Conflict Orientation Modulates R-Loop Levels and Activates Distinct DNA Damage Responses

Published:2017-08 Issue:4 Volume:170 Page:774-786.e19
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Hamperl Stephan,Bocek Michael J.,Saldivar Joshua C.,Swigut Tomek,Cimprich Karlene A.

Funder

NIH

American Cancer Society

Burroughs Wellcome Fund

German Research Foundation DFG

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference73 articles.

1. R loops: from transcription byproducts to threats to genome stability;Aguilera;Mol. Cell,2012

2. The contribution of dormant origins to genome stability: from cell biology to human genetics;Alver;DNA Repair (Amst.),2014

3. Senataxin associates with replication forks to protect fork integrity across RNA-polymerase-II-transcribed genes;Alzu;Cell,2012

4. HTSeq--a Python framework to work with high-throughput sequencing data;Anders;Bioinformatics,2015

5. Nuclear stability and transcriptional directionality separate functionally distinct RNA species;Andersson;Nat. Commun.,2014

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2. Transcription reprogramming and endogenous DNA damage;DNA Repair;2024-10

3. RAD52 resolves transcription-replication conflicts to mitigate R-loop induced genome instability;Nature Communications;2024-09-05

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