A Brucella Type IV Effector Targets the COG Tethering Complex to Remodel Host Secretory Traffic and Promote Intracellular Replication-Reference-Cited by-同舟云学术

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A Brucella Type IV Effector Targets the COG Tethering Complex to Remodel Host Secretory Traffic and Promote Intracellular Replication

Published:2017-09 Issue:3 Volume:22 Page:317-329.e7
ISSN:1931-3128
Container-title:Cell Host & Microbe
language:en
Short-container-title:Cell Host & Microbe

Author:

Miller Cheryl N.,Smith Erin P.,Cundiff Jennifer A.,Knodler Leigh A.,Bailey Blackburn Jessica,Lupashin Vladimir,Celli Jean

Funder

NIH

Washington State University funds

Paul G. Allen School for Global Animal Health

USDA NIFA

USDA NIFA postdoctoral fellowship

Publisher

Elsevier BV

Subject

Virology,Microbiology,Parasitology

Reference49 articles.

1. Contrasting roles of macrophages and dendritic cells in controlling initial pulmonary Brucella infection;Archambaud;Eur. J. Immunol.,2010

2. Interactions of the human pathogenic Brucella species with their hosts;Atluri;Annu. Rev. Microbiol.,2011

3. COG complex complexities: detailed characterization of a complete set of HEK293T cells lacking individual COG subunits;Bailey Blackburn;Front. Cell Dev. Biol.,2016

4. Brucella evades macrophage killing via VirB-dependent sustained interactions with the endoplasmic reticulum;Celli;J. Exp. Med.,2003

5. Brucella coopts the small GTPase Sar1 for intracellular replication;Celli;Proc. Natl. Acad. Sci. USA,2005

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