miR-122 Stimulates Hepatitis C Virus RNA Synthesis by Altering the Balance of Viral RNAs Engaged in Replication versus Translation-Reference-Cited by-同舟云学术

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miR-122 Stimulates Hepatitis C Virus RNA Synthesis by Altering the Balance of Viral RNAs Engaged in Replication versus Translation

Published:2015-02 Issue:2 Volume:17 Page:217-228
ISSN:1931-3128
Container-title:Cell Host & Microbe
language:en
Short-container-title:Cell Host & Microbe

Author:

Masaki Takahiro,Arend Kyle C.,Li You,Yamane Daisuke,McGivern David R.,Kato Takanobu,Wakita Takaji,Moorman Nathaniel J.,Lemon Stanley M.

Publisher

Elsevier BV

Subject

Virology,Microbiology,Parasitology

Reference40 articles.

1. Translating ribosomes inhibit poliovirus negative-strand RNA synthesis;Barton;J. Virol.,1999

2. miR-122, a mammalian liver-specific microRNA, is processed from hcr mRNA and may downregulate the high affinity cationic amino acid transporter CAT-1;Chang;RNA Biol.,2004

3. MicroRNA-122 dependent binding of Ago2 protein to hepatitis C virus RNA is associated with enhanced RNA stability and translation stimulation;Conrad;PLoS ONE,2013

4. Sequences in the 5′ nontranslated region of hepatitis C virus required for RNA replication;Friebe;J. Virol.,2001

5. Interaction of poly(rC)-binding protein 2 with the 5′-terminal stem loop of the hepatitis C-virus genome;Fukushi;Virus Res.,2001

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