Rare splicing defects of FAS underly severe recessive autoimmune lymphoproliferative syndrome-Reference-Cited by-同舟云学术

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Rare splicing defects of FAS underly severe recessive autoimmune lymphoproliferative syndrome

Published:2017-10 Issue: Volume:183 Page:17-23
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Agrebi N.,Ben-Mustapha I.,Matoussi N.,Dhouib N.,Ben-Ali M.,Mekki N.,Ben-Ahmed M.,Larguèche B.,Ben Becher S.,Béjaoui M.,Barbouche M.R.

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference36 articles.

1. What is apoptosis, and why is it important?;Renehan;BMJ,2001

2. Mature T lymphocyte apoptosis--immune regulation in a dynamic and unpredictable antigenic environment;Lenardo;Annu. Rev. Immunol.,1999

3. Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor;Kischkel;EMBO J.,1995

4. FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death inducing signaling complex;Muzio;Cell,1996

5. Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death;Boldin;Cell,1996

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