Cbl-b-deficient mice express alterations in trafficking-related molecules but retain sensitivity to the multiple sclerosis therapeutic agent, FTY720-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Cbl-b-deficient mice express alterations in trafficking-related molecules but retain sensitivity to the multiple sclerosis therapeutic agent, FTY720

Published:2015-05 Issue:1 Volume:158 Page:103-113
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Fujiwara Mai,Anstadt Emily J.,Khanna Kamal M.,Clark Robert B.

Funder

National Multiple Sclerosis Society

National Institute of Health

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference47 articles.

1. Risk alleles for multiple sclerosis identified by a genomewide study;International Multiple Sclerosis Genetics, C.;N. Engl. J. Med.,2007

2. Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis;International Multiple Sclerosis Genetics, C.;Nature,2011

3. Variants within the immunoregulatory CBLB gene are associated with multiple sclerosis;Sanna;Nat. Genet.,2010

4. Negative regulation of lymphocyte activation and autoimmunity by the molecular adaptor Cbl-b;Bachmaier;Nature,2000

5. Cbl-b regulates the CD28 dependence of T-cell activation;Chiang;Nature,2000

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Recycling and Reshaping—E3 Ligases and DUBs in the Initiation of T Cell Receptor-Mediated Signaling and Response;International Journal of Molecular Sciences;2022-03-22

2. Deubiquitylating enzymes: potential target in autoimmune diseases;Inflammopharmacology;2021-11-18

3. Therapeutic Value of Single Nucleotide Polymorphisms on the Efficacy of New Therapies in Patients with Multiple Sclerosis;Journal of Personalized Medicine;2021-04-23

4. Spatiotemporal resolution of spinal meningeal and parenchymal inflammation during experimental autoimmune encephalomyelitis;Neurobiology of Disease;2017-12

5. Cbl-b Deficiency Mediates Resistance to Programed Death-Ligand 1/Programed Death-1 Regulation;Frontiers in Immunology;2017-01-26

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3