OAS1 is not associated with MHC class I hyperexpression in the islets of donors with T1D regardless of disease duration-Reference-Cited by-同舟云学术

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OAS1 is not associated with MHC class I hyperexpression in the islets of donors with T1D regardless of disease duration

Published:2018-06 Issue: Volume:191 Page:34-36
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Sabouri Somayeh,Mikulski Zbigniew^ORCID,von Herrath Matthias G.

Funder

La Jolla Institute for Allergy and Immunology

Juvenile Diabetes Research Foundation

National Institutes of Health

National Institute of Allergy and Infectious Diseases

NIDDK

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference6 articles.

1. The viral paradigm in type 1 diabetes: who are the main suspects?;Rodriguez-Calvo;Autoimmun. Rev.,2016

2. Viral encounters with 2′,5′-oligoadenylate synthetase and RNase L during the interferon antiviral response;Silverman;J. Virol.,2007

3. Oligoadenylate synthase-like (OASL) proteins: dual functions and associations with diseases;Choi;Exp. Mol. Med.,2015

4. Interferon stimulates the expression of 2′,5′-oligoadenylate synthetase and MHC class I antigens in insulin-producing cells;Bonnevie-Nielsen;J. Interf. Res.,1991

5. The antiviral 2′,5′-oligoadenylate synthetase is persistently activated in type 1 diabetes;Bonnevie-Nielsen;Clin. Immunol.,2000

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1. Screening key genes and signaling pathways in COVID-19 infection and its associated complications by integrated bioinformatics analysis;2021-09-25

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