Peptidase inhibitor 16 promotes inflammatory arthritis by suppressing Foxp3 expression via regulating K48-linked ubiquitin degradation Bmi-1 in regulatory T cells-Reference-Cited by-同舟云学术

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Peptidase inhibitor 16 promotes inflammatory arthritis by suppressing Foxp3 expression via regulating K48-linked ubiquitin degradation Bmi-1 in regulatory T cells

Published:2024-02 Issue: Volume:259 Page:109883
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Wang Fang,Gu Xin,Lin Shiyu,Wu Qin,Sun Yuankai,Zhang Qian,Luo Aishu,Feng Xiaoke,Wang Lei,Xu Lingxiao,Sun Wei,Tan Wenfeng

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference48 articles.

1. Network meta-analysis of tofacitinib versus biologic treatments in moderate-to-severe rheumatoid arthritis patients;Camean-Castillo;J. Clin. Pharm. Ther.,2019

2. Rheumatoid arthritis;Smolen;Lancet,2016

3. Rebalancing immune homeostasis to treat autoimmune diseases;Horwitz;Trends Immunol.,2019

4. Tregs and rheumatoid arthritis;Anderson;Acta Reumatol. Port.,2008

5. Induced regulatory T cells: their development, stability, and applications;Kanamori;Trends Immunol.,2016

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Regulatory role of PI16 in autoimmune arthritis and intestinal inflammation: implications for Treg cell differentiation and function;Journal of Translational Medicine;2024-04-02

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