Fingolimod induces BAFF and expands circulating transitional B cells without activating memory B cells and plasma cells in multiple sclerosis-Reference-Cited by-同舟云学术

Fingolimod induces BAFF and expands circulating transitional B cells without activating memory B cells and plasma cells in multiple sclerosis

Published:2018-02 Issue: Volume:187 Page:95-101
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Miyazaki Yusei,Niino Masaaki,Takahashi Eri,Suzuki Masako,Mizuno Masanori,Hisahara Shin,Fukazawa Toshiyuki,Amino Itaru,Nakano Fumihito,Nakamura Masakazu,Akimoto Sachiko,Minami Naoya,Fujiki Naoto,Doi Shizuki,Shimohama Shun,Terayama Yasuo,Kikuchi Seiji

Funder

Ministry of Health, Labor and Welfare of Japan

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference43 articles.

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2. Fingolimod in multiple sclerosis: mechanisms of action and clinical efficacy;Ingwersen;Clin. Immunol.,2012

3. Suppressed pro-inflammatory properties of circulating B cells in patients with multiple sclerosis treated with fingolimod, based on altered proportions of B-cell subpopulations;Miyazaki;Clin. Immunol.,2014

4. Compositional changes of B and T cell subtypes during fingolimod treatment in multiple sclerosis patients: a 12-month follow-up study;Claes;PLoS One,2014

5. Differential effects of fingolimod on B-cell populations in multiple sclerosis;Nakamura;Mult. Scler.,2014

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1. S1PR4 deficiency results in reduced germinal center formation but only marginally affects antibody production;Frontiers in Immunology;2022-12-02

2. Dysregulation of Gene Expressions in Multiple Sclerosis: TNFSF13B and Other Candidate Genes;Journal of Integrative Neuroscience;2022-11-25

3. Proportions of circulating transitional B cells associate with MRI activity in interferon beta-treated multiple sclerosis patients;Journal of Neuroimmunology;2021-09

4. Treatment-Induced BAFF Expression and B Cell Biology in Multiple Sclerosis;Frontiers in Immunology;2021-05-26

5. B-cells expressing NgR1 and NgR3 are localized to EAE-induced inflammatory infiltrates and are stimulated by BAFF;Scientific Reports;2021-02-03