Metabolomic basis for response to high dose vitamin D in critical illness-Reference-Cited by-同舟云学术

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Metabolomic basis for response to high dose vitamin D in critical illness

Published:2021-04 Issue:4 Volume:40 Page:2053-2060
ISSN:0261-5614
Container-title:Clinical Nutrition
language:en
Short-container-title:Clinical Nutrition

Author:

Amrein Karin,Lasky-Su Jessica A.,Dobnig Harald,Christopher Kenneth B.

Funder

Foundation for the National Institutes of Health

Publisher

Elsevier BV

Subject

Critical Care and Intensive Care Medicine,Nutrition and Dietetics

Reference47 articles.

1. Metabolism, metabolome, and metabolomics in intensive care: is it time to move beyond monitoring of glucose and lactate?;Kiehntopf;Am J Respir Crit Care Med,2013

2. Integrative “omic” analysis of experimental bacteremia identifies a metabolic signature that distinguishes human sepsis from systemic inflammatory response syndromes;Langley;Am J Respir Crit Care Med,2014

3. Effect of high-dose vitamin D3 on hospital length of stay in critically ill patients with vitamin D deficiency: the VITdAL-ICU randomized clinical trial;Amrein;JAMA,2014

4. Metabolome alterations in severe critical illness and vitamin D status;Lasky-Su;Crit Care,2017

5. Patterns of genome-wide VDR locations;Tuoresmaki;PloS One,2014

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1. New randomized controlled trials on micronutrients in critical care nutrition: A narrative review;Nutrition in Clinical Practice;2024-08-09

2. High-resolution plasma metabolomics and thiamine status in critically Ill adult patients;Metabolomics;2024-07-27

3. Standard Doses of Cholecalciferol Reduce Glucose and Increase Glutamine in Obesity-Related Hypertension: Results of a Randomized Trial;International Journal of Molecular Sciences;2024-03-18

4. Low plasma vitamin D is associated with increased 28-day mortality and worse clinical outcomes in critically ill patients;BMC Nutrition;2024-01-09

5. General adaptation in critical illness 2: The glucocorticoid signaling system as a master rheostat of homeostatic corrections in concerted action with mitochondrial and essential micronutrient support;Stress: Immunology and Inflammation;2024

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