Content of adenosine 3′:5′-cyclic monophosphate in the pancreatic islets of mice with a hereditary defect of insulin secretion
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference12 articles.
1. Studies with the mutation, diabetes, in the mouse
2. The influence of genetic background on expression of mutations at the diabetes locus in the mouse. I. C57BL/KsJ and C57BL/6J strains
3. INFLUENCE OF THE MUTATION "DIABETES" ON INSULIN RELEASE AND ISLET MORPHOLOGY IN MICE OF DIFFERENT GENETIC BACKGROUNDS
4. The mode of action of adenosine 3′:5′-cyclic monophosphate in mammalian islets of Langerhans. Effects of insulin secretagogues on islet-cell protein kinase activity
5. Concentration of adenosine 3′:5′-cyclic monophosphate in mouse pancreatic islets measured by a protein-binding radioassay
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1. THE ENDOCRINE PANCREAS IN EARLY ALLOXAN DIABETES;Acta Pathologica Microbiologica Scandinavica Section A Pathology;2009-08-15
2. Single gene effect on alcohol preference in the Zucker rats;Pharmacology Biochemistry and Behavior;1994-03
3. Defective regulation of insulin secretion in diabetes and insulinoma;Biochemical Society Transactions;1990-02-01
4. The dynamic insulin secretory response of isolated pancreatic islets of the diabetic mouse. Evidence for a gene dosage effect on insulin secretion;Diabetes;1984-11-01
5. A Temporal Study of Somatostatin Secretion and Its Inhibitory Effect in Genetically Diabetic Mice (C57BL/ KsJ-db/db)*;Endocrinology;1983-12
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