Nitrendipine potentiates bay K 8644-induced contraction of isolated porcine coronary artery: Evidence for functionally distinct dihydropyridine receptor subtypes-Reference-Cited by-同舟云学术

Nitrendipine potentiates bay K 8644-induced contraction of isolated porcine coronary artery: Evidence for functionally distinct dihydropyridine receptor subtypes

Published:1985-05 Issue:3 Volume:128 Page:1295-1302
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Dube Gregory P.,Baik Yung H.,Vaghy Pal L.,Schwartz Arnold

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference28 articles.

1. Novel dihydropyridines with positive inotropic action through activation of Ca2+ channels

2. Correlation of nitrendipine and bay k 8644 binding to isolated canine heart sarcolemma with their pharmacological effects on the canine heart

3. Effects of Bay k 8644, a dihydropyridine analog, on [3H]nitrendipine binding to canine cardiac sarcolemma and the relationship to a positive inotropic effect.

4. Differential effects of Bay k 8644, a presumed calcium channel activator, on sinoatrial nodal and ventricular automaticity of the dog heart

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1. Pharmacologic and radioligand binding analysis of the actions of 1,4-dihydropyridine activators related to Bay K 8644 in smooth muscle, cardiac muscle and neuronal preparations;Naunyn-Schmiedeberg's Archives of Pharmacology;2004

2. Effects of dihydropyridine Ca2+ channel blockers on the discriminative stimulus and the motor impairing effects of (±)-Bay K 8644;European Journal of Pharmacology;1997-10

3. Calcium entry and myogenic phenomena in skeletal muscle arterioles;American Journal of Physiology-Heart and Circulatory Physiology;1994-09-01

4. Regulation and modulation of calcium channels in cardiac, skeletal, and smooth muscle cells;Physiological Reviews;1994-04-01

5. Molecular localization of regions in the L-type calcium channel critical for dihydropyridine action;Neuron;1993-12