Pertussis toxin inhibits EGF-, phorbol ester- and insulin-stimulated DNA synthesis in BALBc3T3 cells: Evidence for post-receptor activation of G iα
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference18 articles.
1. Molecular analysis of signal transduction by growth factors
2. Reduction of epidermal growth factor receptor affinity by heterologous ligands: Evidence for a mechanism involving the breakdown of phosphoinositides and the activation of protein kinase C
3. Early events elicited by bombesin and structurally related peptides in quiescent Swiss 3T3 cells. I. Activation of protein kinase C and inhibition of epidermal growth factor binding.
4. Serum, bradykinin and vasopressin stimulate release of inositol phosphates from human fibroblasts
5. Pertussis toxin inhibits thrombin-induced activation of phosphoinositide hydrolysis and Na+/H+ exchange in hamster fibroblasts.
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2. Positive cooperativity between the thrombin and bradykinin B2receptors enhances arachidonic acid release;American Journal of Physiology-Heart and Circulatory Physiology;2006-03
3. Exogenous myristoylated-Gi2α subunits of GTP-binding proteins are mitogens following their internalization by astrocytes in culture;Molecular Brain Research;2003-01
4. Specific involvement of Gαi2 with epidermal growth factor receptor signaling in rat hepatocytes, and the inhibitory effect of chronic ethanol22Abbreviations: EGFR, epidermal growth factor receptor; PLC-γ1, phospholipase C-γ1; Gαi, inhibitory guanine nucleotide-binding regulatory protein; [Ca2+]i, intracellular calcium concentration; PMSF, phenylmethylsulfonylfluoride.;Biochemical Pharmacology;2001-04
5. Activation of Endogenous Thrombin Receptors Causes Clustering and Sensitization of Epidermal Growth Factor Receptors of Swiss 3t3 Cells without Transactivation;Journal of Cell Biology;2001-01-22
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