Evidence that the lipolytic defect induced by estradiol-treatment in hamster adipocytes is related to an estrogen receptor-mediated defect in the adenylate cyclase catalytic subunit but not in NS-Reference-Cited by-同舟云学术

Evidence that the lipolytic defect induced by estradiol-treatment in hamster adipocytes is related to an estrogen receptor-mediated defect in the adenylate cyclase catalytic subunit but not in NS

Published:1987-05 Issue:1 Volume:145 Page:369-375
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Pecquery René,Leneveu Marie-Christine,Giudicelli Yves

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

1. Estradiol Treatment Decreases the Lipolytic Responses of Hamster White Adipocytes through a Reduction in the Activity of the Adenylate Cyclase Catalytic Subunit*

2. The role of hormone receptors and GTP-regulatory proteins in membrane transduction

3. [3H]GDP release from rat and hamster adipocyte membranes independently linked to receptors involved in activation or inhibition of adenylate cyclase. Differential susceptibility to two bacterial toxins.

4. Inhibition by estrogen of autofeedback regulation of prolactin secretion

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