Mobilization of mitochondrial Ca2+ by hydroperoxy-eicosatetraenoic acid
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference19 articles.
1. Release of Ca2+ from a nonmitochondrial intracellular store in pancreatic acinar cells by inositol-1,4,5-trisphosphate
2. Effect of inositol-1,4,5-trisphosphate on isolated subcellular fractions of rat pancreas
3. Hydroperoxide-induced loss of pyridine nucleotides and release of calcium from rat liver mitochondria.
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2. 12(S)-Hydroperoxyeicosatetraenoic acid (12-HETE) increases mitochondrial nitric oxide by increasing intramitochondrial calcium;Archives of Biochemistry and Biophysics;2007-12
3. Ca2+ signal stimulates the expression of steroidogenic acute regulatory protein and steroidogenesis in bovine adrenal fasciculata–reticularis cells;Life Sciences;2006-05
4. Calcium ion as a second messenger for o-nitrophenylsulfenyl-adrenocorticotropin (NPS-ACTH) and ACTH in bovine adrenal steroidogenesis;ENDOCRINOLOGY;1998
5. Calcium Ion as a Second Messenger for o-Nitrophenylsulfenyl-Adrenocorticotropin (NPS-ACTH) and ACTH in Bovine Adrenal Steroidogenesis**This work was supported, in part, by a Grant-in-Aid for Scientific Research from the Ministry of Education, Science and Culture of Japan (09680623, Priority Area 09235223).;Endocrinology;1998-12-01
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