Differential role of TNF-α and IFN-γ in the brain of rats with chronic relapsing autoimmune encephalomyelitis

Author:

Tanuma Naoyuki,Shin Taekyun,Kogure Kazunari,Matsumoto Yoh

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference26 articles.

1. Enhancement of experimental allergic encephalomyelitis in mice by antibodies against IFN-γ;Billiau;J. Immunol.,1988

2. Staphylococcal enterotoxin B and tumor necrosis factor α-induced relapses of experimental allergic encephalomyelitis: protection by transforming growth factor-β and interleukin-10;Crisi;Eur. J. Immunol.,1995

3. Effect of anti-interferon-γ monoclonal antibody treatment on the development of experimental allergic encephalomyelitis in resistant mouse strains;Duong;J. Neuroimmunol.,1994

4. Tumor necrosis factor-α and lymphotoxin-α are not required for induction of acute experimental autoimmune encephalomyelitis;Frei;J. Exp. Med.,1997

5. Regulation by interferons of the local inflammatory response to bacterial lipopolysaccharide;Heremans;J. Immunol.,1987

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