T-cells specific for soluble recombinant oligodendrocyte-specific protein induce severe clinical experimental autoimmune encephalomyelitis in H-2b and H-2s mice-Reference-Cited by-同舟云学术

T-cells specific for soluble recombinant oligodendrocyte-specific protein induce severe clinical experimental autoimmune encephalomyelitis in H-2b and H-2s mice

Published:2000-06 Issue:1 Volume:105 Page:39-45
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Zhong Ming-Chao,Cohen Lydia,Meshorer Asher,Kerlero de Rosbo Nicole,Ben-Nun Avraham

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference12 articles.

1. Oligodendrocyte-specific protein (OSP) is a major component of CNS myelin;Bronstein;J. Neurosci. Res.,1997

2. Isolation and characterization of a novel oligodendrocyte-specific protein;Bronstein;Neurology,1996

3. The central nervous system-specific myelin oligodendrocytic basic protein (MOBP) is encephalitogenic and a potential target antigen in multiple sclerosis (MS);Kaye;J. Neuroimmunol.,2000

4. T-cell responses to myelin antigens in multiple sclerosis. Relevance of the predominant autoimmune reactivity to myelin oligodendrocyte glycoprotein;Kerlero de Rosbo;J. Autoimmun.,1998

5. Experimental autoimmune encephalomyelitis induced by various antigens of the central nervous system. Overview and relevance to multiple sclerosis;Kerlero de Rosbo,1999

Cited by 34 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Autoimmune Models;Reference Module in Biomedical Sciences;2024

2. Animal Models of Multiple Sclerosis;Current Protocols;2021-06

3. Circulating Picomolar Levels of CCL2 Downregulate Ongoing Chronic Experimental Autoimmune Encephalomyelitis by Induction of Regulatory Mechanisms;The Journal of Immunology;2019-09-04

4. Modulation of MS-like disease by a multi epitope protein is mediated by induction of CD11c+CD11b+Gr1+ myeloid-derived dendritic cells;Journal of Neuroimmunology;2019-08

5. Autoimmune Models;Comprehensive Toxicology;2018