Dinucleotide repeat expansion in the CTLA-4 gene leads to T cell hyper-reactivity via the CD28 pathway in myasthenia gravis
Author:
Publisher
Elsevier BV
Subject
Clinical Neurology,Neurology,Immunology,Immunology and Allergy
Reference64 articles.
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1. Identification of the regulatory role of lncRNA HCG18 in myasthenia gravis by integrated bioinformatics and experimental analyses;Journal of Translational Medicine;2021-11-18
2. Suppression of the CD28/B7 pathway reduces the occurrence and development of myasthenia gravis and cytokine levels;International Journal of Neuroscience;2020-05-18
3. Association between CTLA-4 gene polymorphism and myasthenia gravis in a Chinese cohort;Journal of Clinical Neuroscience;2019-11
4. Autoimmune Myasthenia Gravis;Genetics of Rare Autoimmune Diseases;2019
5. CTLA-4 methylation regulates the pathogenesis of myasthenia gravis and the expression of related cytokines;Medicine;2018-05
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