Tumor necrosis factor-α, lymphotoxin, interleukin (IL)-6, IL-10, IL-12 and perforin mRNA expression in mononuclear cells in response to acetylcholine receptor is augmented in myasthenia gravis

Author:

Matusevicius Darius,Navikas Vaidrius,Palasik Witold,Pirskanen Ritva,Fredrikson Sten,Link Hans

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference39 articles.

1. Defective T lymphocyte function in nonthymectomized patients with myasthenia gravis;Ahlberg;Clin. Immunol. Immunopath.,1991

2. Insulin-dependent diabetes in the NOD mouse model;Anderson;Autoimmunity,1993

3. Cooperation of natural cell stimulatory factor/interleukin-12 with other stimuli in the induction of cytokines and cytotoxic cell-associated molecules in human T and NK cells;Aste-Amezaga;Cell. Immunol.,1994

4. Interleukin-6 overproduction by cultured thymic epithelial cells from patients with myasthenia gravis is potentially involved in thymic hyperplasia;Cohen-Kaminsky;Eur. Cytokine Netw.,1993

5. Immune activation in myasthenia gravis: soluble interleukin-2 receptor, interferon-γ and tumor necrosis factor-α levels in patients' serum;Confalonieri;J. Neuroimmunol.,1993

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