Acute and relapsing experimental autoimmune encephalomyelitis are regulated by differential expression of the CC chemokines macrophage inflammatory protein-1α and monocyte chemotactic protein-1

Author:

Kennedy Kevin J.,Strieter Robert M.,Kunkel Steven L.,Lukacs Nicholas W.,Karpus William J.

Publisher

Elsevier BV

Subject

Clinical Neurology,Neurology,Immunology,Immunology and Allergy

Reference44 articles.

1. Relevance of experimental allergic encephalomyelitis to multiple sclerosis;Arnason;Neurol. Clin.,1983

2. Surface expression of alpha 4 integrin by CD4 T cells is required for their entry into brain parenchyma;Baron;J. Exp. Med.,1993

3. The effects of macrophage depletion on the clinical and pathologic expression of experimental allergic encephalomyelitis;Brosnan;J. Immunol.,1981

4. The C–C chemokine MCP-1 differentially modulates the avidity of β1 and β2 integrins on T lymphocytes;Carr;Immunity,1996

5. Homing to central nervous system vasculature by antigen-specific lymphocytes: I. Localization of 14C-labeled cells during acute, chronic, and relapsing experimental allergic encephalomyelitis;Cross;Lab. Invest.,1990

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