Differential recognition of MBP epitopes in BALB/c mice determines the site of inflammatory disease induction

Author:

Yoshizawa Izumi,Bronson Roderick,Ben-Nun Avi,Richert John R.,Dorf Martin E.,Abromson-Leeman Sara

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference31 articles.

1. T cell responses to myelin basic protein in experimental autoimmune encephalomyelitis-resistant BALB/c mice;Abromson-Leeman;J. Neuroimmunol.,1993

2. Experimental autoimmune encephalomyelitis-resistant mice have highly encephalitogenic myelin basic protein (MBP)-specific T-cell clones that recognize a MBP peptide with high affinity for MHC Class II;Abromson-Leeman;J. Immunol.,1995

3. Experimental autoimmune peripheral neuritis induced in BALB/c mice by myelin basic protein-specific T-cell clones;Abromson-Leeman;J. Exp. Med.,1995

4. Accumulation of the four myelin basic proteins in the mouse brain during development;Barbarese;J. Neurochem.,1978

5. Detection of autoimmune cells proliferating to myelin basic protein and selection of T cell lines that mediate experimental autoimmune encephalomyelitis (EAE) in mice;Ben-Nun;J. Immunol.,1983

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