Nitric oxide is a potential down-regulating molecule in autoimmune disease: inhibition of nitric oxide production renders PVG rats highly susceptible to EAE

Author:

Cowden W.B,Cullen F.A,Staykova M.A,Willenborg D.O

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference43 articles.

1. Suppression of experimental allergic encephalomyelitis by interferon;Abreu;Immunol. Commun.,1982

2. Nitric oxide control of steroidogenesis—endocrine effects of NG-nitro-l-arginine and comparisons to alcohol;Adams;Life Sci.,1992

3. Suppression of lymphocyte proliferation through the nitric oxide synthesizing pathway;Albina;J. Surg. Res.,1991

4. Nitric oxide production is required for murine resident peritoneal macrophages to suppress mitogen-stimulated T cell proliferation—role of IFN-γ in the induction of the nitric oxide-synthesizing pathway;Albina;J. Immunol.,1991

5. Experimental autoimmune encephalomyelitis (EAE) in genetically resistant rats: PVG rats resist active induction of EAE but are susceptible to and can generate EAE effector T cell lines;Ben Nun;J. Immunol.,1982

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