Copper Chelation via beta-alanine extends lifespan in a C. elegans model of Alzheimer's Disease-Reference-Cited by-同舟云学术

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Copper Chelation via beta-alanine extends lifespan in a C. elegans model of Alzheimer's Disease

Published:2023-06 Issue: Volume:10 Page:100076
ISSN:2666-4593
Container-title:Brain Disorders
language:en
Short-container-title:Brain Disorders

Author:

Pulido Arnulfo,Hulbert Benjamin,Giese Hayleigh,Kurian Sabrina,Rozhon Rebbeca,Zambrano Michael,Diaz Oscar,Abd Mariam,Caputo Madison,Kissel Daniel S.,Havens Mallory A.^ORCID

Publisher

Elsevier BV

Subject

General Earth and Planetary Sciences,General Environmental Science

Reference49 articles.

1. Emmady, P.D. and P. Tadi, Dementia, in StatPearls. 2022: Treasure Island (FL).

2. The amyloid hypothesis of Alzheimer's disease at 25 years;Selkoe;EMBO Mol. Med.,2016

3. Alzheimer's disease: pathogenesis, diagnostics, and therapeutics;Tiwari;Int J Nanomedicine,2019

4. Role of mitochondrial amyloid-beta in Alzheimer's disease;Chen;J. Alzheimers Dis.,2010

5. Contributing Factors of Neurodegeneration in Alzheimer's Disease, in Alzheimer's Disease;Tailor,2019

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Omics analysis unveils changes in the metabolome and lipidome of Caenorhabditis elegans upon polydopamine exposure;Journal of Pharmaceutical and Biomedical Analysis;2024-07

2. Copper: From enigma to therapeutic target for neurological disorder;Basic & Clinical Pharmacology & Toxicology;2024-04-15

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