Analysis of cysteinyl leukotrienes and their metabolites in bile of patients with peroxisomal or mitochondrial β-oxidation defects
Author:
Publisher
Elsevier BV
Subject
Biochemistry, medical,Clinical Biochemistry,Biochemistry,General Medicine
Reference12 articles.
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2. Leukotrierne E4 elimination and metabolism in normal human subjects;Sala;J. Biol. Chem.,1990
3. Peroxisomal degradation of leukotrienes by β-oxidation from the ω-end;Jedlitschky;J. Biol. Chem.,1991
4. Impaired degradation of leukotrienes in patients with peroxisome deficiency disorders;Mayatepek;J. Clin. Invest.,1993
5. Increased excretion of endogenous urinary leukotriene E4 in extrahepatic cholestasis;Mayatepek;Clin. Chim. Acta,1993
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1. Cellular Source of Cysteinyl Leukotrienes Following Chlorine Exposure;American Journal of Respiratory Cell and Molecular Biology;2020-11
2. “Role of peroxisomes in human lipid metabolism and its importance for neurological development”;Neuroscience Letters;2017-01
3. CysLT1 Receptor Is Protective against Oxidative Stress in a Model of Irritant-Induced Asthma;The Journal of Immunology;2016-05-25
4. Peroxisomes in Human Health and Disease: Metabolic Pathways, Metabolite Transport, Interplay with Other Organelles and Signal Transduction;Peroxisomes and their Key Role in Cellular Signaling and Metabolism;2013
5. Leukotriene Receptors;Chemical Reviews;2011-04-28
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