In vivo up-regulation of aryl hydrocarbon receptor expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in a dioxin-resistant rat model☆1☆Portions of this work were presented at the 20th International Symposium on Halogenated Environmental Organic Pollutants & POPS, Monterey, California (August 13–17, 2000).1Abbreviations: AHR, aryl hydrocarbon receptor; AP, alkaliSne phosphatase; ARNT, aryl hydrocarbon receptor nuclear translocator protein; dNTP, 2′-deoxynucleoside 5′-triphosphate; DRE, dioxin responsive element; H/W, Han/Wistar (Kuopio); L-E, Long-Evans (Turku AB); PCB, polychlorinated biphenyl; PCR, polymerase chain reaction; RT, reverse transcription; SD, Sprague Dawley; and TCDD, 2,3,7,8-tetrachlorodibenzo-pdioxin.
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Published:2001-12
Issue:12
Volume:62
Page:1565-1578
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ISSN:0006-2952
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Container-title:Biochemical Pharmacology
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language:en
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Short-container-title:Biochemical Pharmacology
Author:
Franc Monique-Andrée,Pohjanvirta Raimo,Tuomisto Jouko,Okey Allan B
Subject
Pharmacology,Biochemistry
Reference54 articles.
1. The Ah receptor signal transduction pathway;Denison,1998
2. Induction of cytochrome P4501A1;Whitlock;Annu Rev Pharmacol Toxicol,1999
3. Role of the aromatic hydrocarbon receptor and [Ah] gene battery in the oxidative stress response, cell cycle control, and apoptosis;Nebert;Biochem Pharmacol,2000
4. Short-term toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in laboratory animals;Pohjanvirta;Pharmacol Rev,1994
5. The Ah receptor;Okey;Toxicol Lett,1994
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