Nifedipine and bay K inhibit contraction independently from their action on calcium channels
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference22 articles.
1. [3H]nitrendipine labelling of the Ca2+ channel in skeletal muscle
2. Solubilization and partial purification of putative calcium channels labelled with [3H]-nimodipine
3. Determination of the molecular size of the nitrendipine-sensitive Ca2+ channel by radiation inactivation
4. Solubilization of the nitrendipine receptor from skeletal muscle transverse tubule membranes Interactions with specific inhibitors of the voltage-dependent Ca2+ channel
5. The 1,4-dihydropyridine receptor associated with the skeletal muscle voltage-dependent Ca2+ channel. Purification and subunit composition.
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1. [Ca2+]i-sensitive, IP3-independent Ca2+ influx in smooth muscle of rat vas deferens revealed by procaine;British Journal of Pharmacology;1993-12
2. The effect of calcium channel antagonists and bay K 8644 on calcium fluxes of malignant hyperpyrexia-susceptible muscle;International Journal of Biochemistry;1993-04
3. Action of heptaminol hydrochloride on contractile properties in frog isolated twitch muscle fibre;British Journal of Pharmacology;1991-11
4. Multiple effects of BAY K 8644 and nifedipine on isolated diaphragmatic fibers in vitro;Journal of Applied Physiology;1991-09-01
5. The dual effect of BAY K 8644 on excitation-contraction coupling in gastric smooth muscle;General Pharmacology: The Vascular System;1991-01
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