Galectin-3 contributes to acute cardiac dysfunction and toxicity by increasing oxidative stress and fibrosis in doxorubicin-treated mice-Reference-Cited by-同舟云学术

Galectin-3 contributes to acute cardiac dysfunction and toxicity by increasing oxidative stress and fibrosis in doxorubicin-treated mice

Published:2023-12 Issue: Volume:393 Page:131386
ISSN:0167-5273
Container-title:International Journal of Cardiology
language:en
Short-container-title:International Journal of Cardiology

Author:

Seropian Ignacio M.,Fontana Estevez Florencia S.,Villaverde Alejo,Cacciagiú Leonardo,Bustos Romina,Touceda Vanessa,Penas Federico,Selser Carolina,Morales Celina,Miksztowicz Verónica,González Germán E.

Funder

Agencia Nacional de Promoción Científica y Tecnológica

Agencia Nacional De Promoción Científica Y Tecnológica

Consejo Nacional de Investigaciones Científicas y Técnicas

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

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5. A human lectin, galectin-3 (epsilon bp/mac-2), stimulates superoxide production by neutrophils;Yamaoka;J. Immunol.,1995

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