The miR-26b/CTGF axis: A promising therapeutic mechanism for managing myocardial fibrosis post myocardial infarction
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Publisher
Elsevier BV
Reference5 articles.
1. MicroRNA-26b relieves inflammatory response and myocardial remodeling of mice with myocardial infarction by suppression of MAPK pathway through binding to PTGS2[J];Ge;Int. J. Cardiol.,2019
2. MicroR-26b targets high mobility group, AT-hook 2 to ameliorate myocardial infarction-induced fibrosis by suppression of cardiac fibroblasts activation[J];Chen;Curr. Neurovasc. Res.,2020
3. MicroRNA-26b inhibits metastasis of osteosarcoma via targeting CTGF and Smad1[J];Duan;Tumour Biol.,2015
4. LncRNA CFRL aggravates cardiac fibrosis by modulating both miR-3113-5p/CTGF and miR-3473d/FN1 axis[J];Cui;iScience,2023
5. miR-30a attenuates cardiac fibrosis in rats with myocardial infarction by inhibiting CTGF[J];Chen;Exp. Ther. Med.,2018
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