Bidirectional effect of serum amyloid A on plaque stability

Author:

Li Bo,Luo Bei-bei,Qin Wei-dong,Liu Hui,Xia Yan-fei,Liu Tong-xiang,Li Jing-tian,Zhang Ming-xiang,Zhang Yun,Zhang Cheng,An Feng-shuang

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference19 articles.

1. Human monocyte-derived macrophages induce collagen breakdown in fibrous caps of atherosclerotic plaques. Potential role of matrix-degrading metalloproteinases and implications for plaque rupture;Shah;Circulation,1995

2. Collagen synthesis in atherosclerosis: too much and not enough;Rekhter;Cardiovasc Res,1999

3. The major acute phase reactants: C-reactive protein, serum amyloid P component and serum amyloid A protein;Steel;Immunol Today,1994

4. Lipoproteins and acute phase response during acute infection. Interrelationships between C-reactive protein and serum amyloid-A protein and lipoproteins;Sammalkorpi;Ann Med,1990

5. Expression of apolipoprotein serum amyloid A mRNA in human atherosclerotic lesions and cultured vascular cells: implications for serum amyloid A function;Meek;Proc Natl Acad Sci U S A,1994

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