LncRNA MALAT1: A potential regulator of autophagy in myocardial ischemia-reperfusion injury
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine
Reference5 articles.
1. Down-regulation of lncRNA MALAT1 reduces cardiomyocyte apoptosis and improves left ventricular function in diabetic rats;Zhang;Int. J. Cardiol.,2016
2. Long noncoding RNAs in patients with acute myocardial infarction;Vausort;Circ. Res.,2014
3. Long non-coding RNA MALAT1 functions as a mediator in cardioprotective effects of fentanyl in myocardial ischemia-reperfusion injury;Zhao;Cell Biol. Int.,2017
4. MiR-204 regulates cardiomyocyte autophagy induced by ischemia-reperfusion through LC3-II;Xiao;J. Biomed. Sci.,2011
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1. CTCF-mediated H3K27me3 enrichment on the LncRNA MALAT1 promoter regulates the cardiomyocytes from I/R-induced apoptosis through targeting miR-26b-5p;Molecular & Cellular Toxicology;2022-10-27
2. TIPE2 protects cardiomyocytes from ischemia‑reperfusion‑induced apoptosis by decreasing cell autophagy via the mTORC1 signaling pathway;Experimental and Therapeutic Medicine;2022-08-04
3. Long non-coding RNA MALAT1 aggravated liver ischemia-reperfusion injury via targeting miR-150-5p/AZIN1;Bioengineered;2022-05-02
4. Interleukin‐7 aggravates myocardial ischaemia/reperfusion injury by regulating macrophage infiltration and polarization;Journal of Cellular and Molecular Medicine;2021-09-28
5. Non-coding RNAs modulate autophagy in myocardial ischemia-reperfusion injury: a systematic review;Journal of Cardiothoracic Surgery;2021-05-22
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