Cancerous hyper-mutagenesis in p53 genes is possibly associated with transcriptional bypass of DNA lesions

Author:

Rodin S.N.,Rodin A.S.,Juhasz A.,Holmquist G.P.

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Genetics,Molecular Biology

Reference53 articles.

1. Mutations of bacteria from virus sensitivity to virus resistance;Luria;Genetics,1943

2. Mutations in the p53 tumor suppressor gene: clues to cancer etiology and molecular pathogenesis;Greenblatt;Cancer Res.,1994

3. Mutator phenotype may be required for multistage carcinogenesis;Loeb;Cancer Res.,1991

4. Hypermutability and silent mutations in human carcinogenesis;Strauss;Semin. Cancer Biol.,1998

5. CpG transition strand asymmetry and hitch-hiking mutations as measures of tumorigenic selection in shaping the p53 mutation spectrum;Rodin;Int. J. Mol. Med.,1998

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