Fasting potentiates diclofenac-induced liver injury via inductions of oxidative/endoplasmic reticulum stresses and apoptosis, and inhibition of autophagy by depleting hepatic glutathione in mice-Reference-Cited by-同舟云学术

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Fasting potentiates diclofenac-induced liver injury via inductions of oxidative/endoplasmic reticulum stresses and apoptosis, and inhibition of autophagy by depleting hepatic glutathione in mice

Published:2024-05 Issue: Volume:187 Page:114624
ISSN:0278-6915
Container-title:Food and Chemical Toxicology
language:en
Short-container-title:Food and Chemical Toxicology

Author:

Kwon Doyoung,Seo Hyeji,Kim Sou Hyun^ORCID,Chung Ki Wung,Lee Jaewon^ORCID,Jung Young-Suk^ORCID

Funder

National Research Foundation of Korea

Publisher

Elsevier BV

Reference56 articles.

1. Diclofenac-induced liver injury: a paradigm of idiosyncratic drug toxicity;Aithal;Expet Opin. Drug Saf.,2004

2. Diclofenac-associated hepatotoxicity: analysis of 180 cases reported to the food and drug administration as adverse reactions;Banks;Hepatology,1995

3. Diclofenac-induced liver injury: a paradigm of idiosyncratic drug toxicity;Boelsterli;Toxicol. Appl. Pharmacol.,2003

4. Induction of hepatic heme oxygenase-1 by diclofenac in rodents: role of oxidative stress and cytochrome P-450 activity;Cantoni;J. Hepatol.,2003

5. Tissue glutathione as a cyst(e)ine reservoir during fasting and refeeding of rats;Cho;J. Nutr.,1981

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Potential treatment of glutathione in bullfrogs with abnormal hepatic lipid metabolism revealed by hepatic lipid metabolism and serum metabolomics analysis;Frontiers in Cellular and Infection Microbiology;2024-08-13

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