Mechanisms and pathophysiological significance of sterile inflammation during acetaminophen hepatotoxicity-Reference-Cited by-同舟云学术

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Mechanisms and pathophysiological significance of sterile inflammation during acetaminophen hepatotoxicity

Published:2020-04 Issue: Volume:138 Page:111240
ISSN:0278-6915
Container-title:Food and Chemical Toxicology
language:en
Short-container-title:Food and Chemical Toxicology

Author:

Jaeschke Hartmut,Ramachandran Anup

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

NIDDK

National Institute of General Medicine

Publisher

Elsevier BV

Subject

Toxicology,General Medicine,Food Science

Reference136 articles.

1. Increased mitochondrial stress and modulation of mitochondrial respiratory enzyme activities in acetaminophen-induced toxicity in mouse macrophage cells;Al-Belooshi;Food Chem. Toxicol.,2010

2. Molecular forms of HMGB1 and keratin-18 as mechanistic biomarkers for mode of cell death and prognosis during clinical acetaminophen hepatotoxicity;Antoine;J. Hepatol.,2012

3. High-mobility group box-1 protein and keratin-18, circulating serum proteins informative of acetaminophen-induced necrosis and apoptosis in vivo;Antoine;Toxicol. Sci.,2009

4. Secretory leukocyte protease inhibitor: a pivotal mediator of anti-inflammatory responses in acetaminophen-induced acute liver failure;Antoniades;Hepatology,2014

5. Source and characterization of hepatic macrophages in acetaminophen-induced acute liver failure in humans;Antoniades;Hepatology,2012

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