Ursolic acid attenuates lipopolysaccharide-induced cognitive deficits in mouse brain through suppressing p38/NF-κB mediated inflammatory pathways-Reference-Cited by-同舟云学术

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Ursolic acid attenuates lipopolysaccharide-induced cognitive deficits in mouse brain through suppressing p38/NF-κB mediated inflammatory pathways

Published:2011-09 Issue:2 Volume:96 Page:156-165
ISSN:1074-7427
Container-title:Neurobiology of Learning and Memory
language:en
Short-container-title:Neurobiology of Learning and Memory

Author:

Wang Yong-Jian,Lu Jun,Wu Dong-mei,Zheng Zi-hui,Zheng Yuan-Lin,Wang Xiao-hui,Ruan Jie,Sun Xiao,Shan Qun,Zhang Zi-feng

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Cognitive Neuroscience,Experimental and Cognitive Psychology

Reference43 articles.

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2. Broad expression of toll-like receptors in the human central nervous system;Bsibsi;Journal of Neuropathology Experimental Neurology,2002

3. Toll-like receptor 4 on nonhematopoietic cells sustains CNS inflammation during endotoxemia, independent of systemic cytokines;Chakravarty;The Journal of Neuroscience,2005

4. Tomatidine inhibits iNOS and COX-2 through suppression of NF-κB and JNK pathways in LPS-stimulated mouse macrophages;Chiu;FEBS Letters,2008

5. LPS induction of I[kappa]B-[alpha] degradation and iNOS expression in a skin dendritic cell line is prevented by the Janus kinase 2 inhibitor, Tyrphostin B42;Cruz;Nitric Oxide,2001

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