HDAC-mediated control of ERK- and PI3K-dependent TGF-β-induced extracellular matrix-regulating genes-Reference-Cited by-同舟云学术

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HDAC-mediated control of ERK- and PI3K-dependent TGF-β-induced extracellular matrix-regulating genes

Published:2010-09 Issue:7 Volume:29 Page:602-612
ISSN:0945-053X
Container-title:Matrix Biology
language:en
Short-container-title:Matrix Biology

Author:

Barter Matt J.,Pybus Leon,Litherland Gary J.,Rowan Andrew D.,Clark Ian M.,Edwards Dylan R.,Cawston Tim E.,Young David A.

Funder

Arthritis Research Campaign

Dunhill Medical Trust

JGW Patterson Foundation

Fighting Arthritis in the North East

Medical Research Council

Publisher

Elsevier BV

Subject

Molecular Biology

Reference47 articles.

1. The selectivity of protein kinase inhibitors: a further update;Bain;Biochem. J.,2007

2. Acetylation of mitogen-activated protein kinase phosphatase-1 inhibits Toll-like receptor signaling;Cao;J. Exp. Med.,2008

3. Duration of nuclear NF-kappaB action regulated by reversible acetylation;Chen;Science,2001

4. Histone acetylation-independent effect of histone deacetylase inhibitors on Akt through the reshuffling of protein phosphatase 1 complexes;Chen;J. Biol. Chem.,2005

5. Snail is required for transforming growth factor-beta-induced epithelial–mesenchymal transition by activating PI3 kinase/Akt signal pathway;Cho;Biochem. Biophys. Res. Commun.,2007

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