Mice that lack matrix metalloproteinase-9 display delayed wound healing associated with delayed reepithelization and disordered collagen fibrillogenesis

Author:

Kyriakides Themis R.,Wulsin Drausin,Skokos Eleni A.,Fleckman Philip,Pirrone Annalisa,Shipley J. Michael,Senior Robert M.,Bornstein Paul

Publisher

Elsevier BV

Subject

Molecular Biology

Reference42 articles.

1. The lack of thrombospondin-1 (TSP1) dictates the course of wound healing in double-TSP1/TSP2-null mice;Agah;Am. J. Pathol.,2002

2. Thrombospondin 2 levels are increased in aged mice: consequences for cutaneous wound healing and angiogenesis;Agah;Matrix Biol.,2004

3. Topical synthetic inhibitor of matrix metalloproteinases delays epidermal regeneration of human wounds;Agren;Exp. Dermatol.,2001

4. Matrix metalloproteinases (MMP9 and MMP2) induce the release of vascular endothelial growth factor (VEGF) by ovarian carcinoma cells: implications for ascites formation;Belotti;Cancer Res.,2003

5. Matrix metalloproteinase-9 triggers the angiogenic switch during carcinogenesis;Bergers;Nat. Cell Biol.,2000

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