Lack of the myotendinous junction marker col22a1 results in posture and locomotion disabilities in zebrafish-Reference-Cited by-同舟云学术

Lack of the myotendinous junction marker col22a1 results in posture and locomotion disabilities in zebrafish

Published:2022-05 Issue: Volume:109 Page:1-18
ISSN:0945-053X
Container-title:Matrix Biology
language:en
Short-container-title:Matrix Biology

Author:

Malbouyres Marilyne,Guiraud Alexandre^ORCID,Lefrançois Christel,Salamito Mélanie^ORCID,Nauroy Pauline,Bernard Laure,Sohm Frédéric^ORCID,Allard Bruno,Ruggiero Florence^ORCID

Funder

AFM-Telethon

FRM

Publisher

Elsevier BV

Subject

Molecular Biology

Reference45 articles.

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3. The zebrafish candyfloss mutant implicates extracellular matrix adhesion failure in laminin 2-deficient congenital muscular dystrophy;Hall;Proc. Natl. Acad. Sci.,2007

4. Dystrophin deficiency is associated with myotendinous junction defects in prenecrotic and fully regenerated skeletal muscle;Law;Am. J. Pathol.,1993

5. Site and mechanics of failure in normal and dystrophin deficient skeletal muscle;Law;Muscle Nerve,1995

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