Bloom’s syndrome: Why not premature aging?
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Neurology,Molecular Biology,Ageing,Biochemistry,Biotechnology
Reference186 articles.
1. Nascent chromatin capture proteomics determines chromatin dynamics during DNA replication and identifies unknown fork components;Alabert;Nat. Cell Biol.,2014
2. Mobile D-loops are a preferred substrate for the Bloom's syndrome helicase;Bachrati;Nucleic Acids Res.,2006
3. The BLM helicase contributes to telomere maintenance through processing of late-replicating intermediate structures;Barefield;Nucleic Acids Res.,2012
4. Mechanisms by which Bloom protein can disrupt recombination intermediates of Okazaki fragment maturation;Bartos;J. Biol. Chem.,2006
5. Chromatid exchanges in ataxia telangiectasia, Bloom syndrome Werner syndrome, and xeroderma pigmentosum;Bartram;Ann. Hum. Genet.,1976
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