Epidermal growth factor activates m-calpain, resulting in apoptosis of HaCaT keratinocytes
Author:
Publisher
Elsevier BV
Subject
Dermatology,Molecular Biology,Biochemistry
Reference10 articles.
1. Calpain: new perspectives in molecular diversity and physiological–pathological involvement;Saido;FASEB J,1994
2. Membrane proximal ERK signaling is required for m-calpain activation downstream of epidermal growth factor receptor signaling;Glading;J. Biol. Chem,2001
3. Matrix-independent survival of human keratinocytes through an EGF receptor/MAPK-kinase dependent pathway;Jost;Mol. Biol. Cell,2001
4. Autolytic transition of μ-calpain upon activation as resolved by antibodies distincting between the pre- and post-autolysis forms;Saido;J. Biochem,1992
5. Simultaneous degradation of αII- and βII-spectrin by caspase 3 (CPP32) in apoptotic cells;Klevin;J. Biol. Chem,1998
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