A novel OSMR mutation in familial primary localized cutaneous amyloidosis in a Japanese family
Author:
Publisher
Elsevier BV
Subject
Dermatology,Molecular Biology,Biochemistry
Reference13 articles.
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2. Amyloidogenesis in organ-limited cutaneous amyloidosis: an antigenic identity between epidermal keratin and skin amyloid;Kobayashi;J Invest Dermatol,1983
3. Oncostatin M receptor-beta mutations underlie familial primary localized cutaneous amyloidosis;Arita;Am J Hum Genet,2008
4. Principles of interleukin (IL)-6-type cytokine signalling and its regulation;Heinrich;Biochem J,2003
5. Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice;Dillon;Nat Immunol,2004
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1. Activating mutations of the gp130/JAK/STAT pathway in human diseases;Advances in Protein Chemistry and Structural Biology;2019
2. Familial primary localized cutaneous amyloidosis in a Japanese family;Journal of Dermatological Science;2016-08
3. Cutaneous Amyloidosis;Current Clinical Pathology;2015
4. Detection of common mutations in sporadic primary localized cutaneous amyloidosis by DNA mass spectrometry;British Journal of Dermatology;2014-04
5. A Novel Missense Mutation in Oncostatin M Receptor Beta Causing Primary Localized Cutaneous Amyloidosis;BioMed Research International;2014
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