Exposure to volatile ferroptosis inhibitor, TEMPO, reduced cutaneous ischemia-reperfusion injury progression to pressure ulcer formation in a mouse model

Author:

Ishikawa Mai,Uchiyama Akihiko,Kosaka Keiji,Nishio Mayu,Ogino Sachiko,Yokoyama Yoko,Torii Ryoko,Akai Ryoko,Iwawaki Takao,Torii Seiji,Motegi Sei-ichiro

Funder

Japan Society for the Promotion of Science

Gunma University

Publisher

Elsevier BV

Reference47 articles.

1. Ischemia-reperfusion injury in chronic pressure ulcer formation: a skin model in the rat;Peirce;Wound Repair Regen.,2000

2. The loss of MCP-1 attenuates cutaneous ischemia-reperfusion injury in a mouse model of pressure ulcer;Saito;J. Invest. Dermatol.,2008

3. Histopathology of pressure ulcers as a result of sequential computer-controlled pressure sessions in a fuzzy rat model;Salcido;Adv. Wound Care,1994

4. Development of a simple, noninvasive, clinically relevant model of pressure ulcers in the mouse;Stadler;J. Invest. Surg.,2004

5. Protective effect of MFG-E8 after cutaneous ischemia-reperfusion injury;Uchiyama;J. Invest. Dermatol.,2015

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