CD22 and CD72 contribute to the development of scleroderma in a murine model-Reference-Cited by-同舟云学术

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CD22 and CD72 contribute to the development of scleroderma in a murine model

Published:2020-01 Issue:1 Volume:97 Page:66-76
ISSN:0923-1811
Container-title:Journal of Dermatological Science
language:en
Short-container-title:Journal of Dermatological Science

Author:

Zhao Chunyan,Matsushita Takashi,Ha Nguyen Vinh Thi,Tennichi Momoko,Fujimoto Manabu,Takehara Kazuhiko,Hamaguchi Yasuhito

Publisher

Elsevier BV

Subject

Dermatology,Molecular Biology,Biochemistry

Reference33 articles.

1. Altered B lymphocyte homeostasis and functions in systemic sclerosis;Forestier;Autoimmun. Rev.,2018

2. Quantitative genetic variation in CD19 expression correlates with autoimmunity in mice and humans;Sato;J. Immunol.,2000

3. CD19-dependent B lymphocyte signaling thresholds influence skin fibrosis and autoimmunity in the tight-skin mouse;Saito;J. Clin. Invest.,2002

4. B-lymphocyte depletion reduces skin fibrosis and autoimmunity in the tight-skin mouse model for systemic sclerosis;Hasegawa;Am. J. Pathol.,2006

5. CD22, a B cell-specific immunoglobulin superfamily member, is a sialic acid-binding lectin;Sgroi;J. Biol. Chem.,1993

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