HLA-B*58:01 strongly associates with allopurinol-induced adverse drug reactions in a Japanese sample population
Author:
Publisher
Elsevier BV
Subject
Dermatology,Molecular Biology,Biochemistry
Reference8 articles.
1. HLA-B*5801 allele as a genetic marker for severe cutaneous adverse reactions caused by allopurinol;Hung;Proc Natl Acad Sci U S A,2005
2. A European study of HLA-B in Stevens–Johnson syndrome and toxic epidermal necrolysis related to five high-risk drugs;Lonjou;Pharmacogenet Genomics,2008
3. A whole-genome association study of major determinants for allopurinol-related Stevens–Johnson syndrome and toxic epidermal necrolysis in Japanese patients;Tohkin;Pharmacogenomics J,2013
4. The spectrum of Stevens–Johnson syndrome and toxic epidermal necrolysis: a clinical classification;Roujeau;J Invest Dermatol,1994
5. HLA-A31 strongly associates with carbamazepine-induced adverse drug reactions but not with carbamazepine-induced lymphocyte proliferation in a Japanese population;Niihara;J Dermatol,2012
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