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N-terminal truncations of substance P1–7 amide affect its action on spinal cord injury-induced mechanical allodynia in rats

  • Published:2014-09 Issue: Volume:738 Page:319-325
  • ISSN:0014-2999
  • Container-title:European Journal of Pharmacology
  • language:en
  • Short-container-title:European Journal of Pharmacology

Author:

Carlsson-Jonsson Anna,Gao Tianle,Hao Jing-Xia,Fransson Rebecca,Sandström Anja,Nyberg Fred,Wiesenfeld-Hallin Zsuzsanna,Xu Xiao-Jun

Funder

Uppsala Berzelii Technology Centre for Neurodiagnostics

Swedish Medical Research Council

Publisher

Elsevier BV

Subject

Pharmacology

Reference33 articles.

1. Dipeptidyl(amino)peptidase IV and aminopeptidase M metabolize circulating substance P in vivo;Ahmad;J. Pharmacol. Exp. Ther.,1992

2. Central pain;Boivie,2006

3. Endomorphin-1 and endomorphin-2 differentially interact with specific binding sites for substance P (SP) aminoterminal SP1-7 in the rat spinal cord;Botros;Peptides,2006

4. Endomorphins interact with the substance P (SP) aminoterminal SP(1-7) binding in the ventral tegmental area of the rat brain;Botros;Peptides,2008

5. Delivery of peptide and protein drugs over the blood-brain barrier;Brasnjevic;Prog. neurobiol.,2009

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