Self-limiting bidirectional positive feedback between P53 and P21 is involved in cardiac hypertrophy-Reference-Cited by-同舟云学术

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Self-limiting bidirectional positive feedback between P53 and P21 is involved in cardiac hypertrophy

Published:2022-10 Issue: Volume:932 Page:175239
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Hong Jia-hui^ORCID,Ding Yuan-Yuan,Li Jing-Mei,Pan Xi-Chun,Liu Ya,Zhang Hai-Gang

Funder

Natural Science Foundation of Chongqing

Third Military Medical University

Publisher

Elsevier BV

Subject

Pharmacology

Reference30 articles.

1. Re-expression of proteins involved in cytokinesis during cardiac hypertrophy;Ahuja;Exp. Cell Res.,2007

2. Length-independent telomere damage drives post-mitotic cardiomyocyte senescence;Anderson;EMBO J.,2019

3. Changes in cardiomyocyte cell cycle and hypertrophic growth during fetal to adult in mammals;Bishop;J. Am. Heart Assoc.,2021

4. Dysregulated phenylalanine catabolism plays a key role in the trajectory of cardiac aging;Czibik;Circulation,2021

5. Cell cycle arrest through indirect transcriptional repression by p53: I have a DREAM;Engeland;Cell Death Differ.,2018

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1. FTO ameliorates doxorubicin-induced cardiotoxicity by inhibiting ferroptosis via P53–P21/Nrf2 activation in a HuR-dependent m6A manner;Redox Biology;2024-04

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