Spinal TASK-1 and TASK-3 modulate inflammatory and neuropathic pain-Reference-Cited by-同舟云学术

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Spinal TASK-1 and TASK-3 modulate inflammatory and neuropathic pain

Published:2019-11 Issue: Volume:862 Page:172631
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

García Guadalupe,Noriega-Navarro Roxana,Martínez-Rojas Vladimir A.,Gutiérrez-Lara Erika J.,Oviedo Norma,Murbartián Janet

Funder

Consejo Nacional de Ciencia y Tecnología

Publisher

Elsevier BV

Subject

Pharmacology

Reference60 articles.

1. TREK2 expressed selectively in IB4-binding C-fiber nociceptors hyperpolarizes their membrane potentials and limits spontaneous pain;Acosta;J. Neurosci.,2014

2. TREK-1, a K+ channel involved in polymodal pain perception;Alloui;EMBO J.,2006

3. Role of opioid receptors in the reduction of formalin-induced secondary allodynia and hyperalgesia in rats;Ambriz-Tututi;Eur. J. Pharmacol.,2009

4. Cellular and molecular mechanisms of pain;Basbaum;Cell,2009

5. Background potassium channel block and TRPV1 activation contribute to proton depolarization of sensory neurons from humans with neuropathic pain;Baumann;Eur. J. Neurosci.,2004

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