Blockade by NNC 55-0396, mibefradil, and nickel of calcium and exocytotic signals in chromaffin cells: Implications for the regulation of hypoxia-induced secretion at early life-Reference-Cited by-同舟云学术

Blockade by NNC 55-0396, mibefradil, and nickel of calcium and exocytotic signals in chromaffin cells: Implications for the regulation of hypoxia-induced secretion at early life

Published:2015-03 Issue: Volume:751 Page:1-12
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Fernández-Morales José C.,Fernando Padín J.,Vestring Stefan,Musial Diego C.,de Diego Antonio-Miguel G.,García Antonio G.

Funder

Ministerio de Economía y Competitividad, Spain

CABICYC; UAM/Bioibérica, Spain

Publisher

Elsevier BV

Subject

Pharmacology

Reference38 articles.

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2. Ca2+-induced Ca2+ release in chromaffin cells seen from inside the ER with targeted aequorin;Alonso;J. Cell Biol.,1999

3. Recent patents on calcium channel blockers: emphasis on CNS diseases;Arranz-Tagarro;Expert Opin. Ther. Pat.,2014

4. Voltage-dependent blockade of diverse types of voltage-gated Ca2+ channels expressed in Xenopus oocytes by the Ca2+ channel antagonist mibefradil (Ro 40-5967);Bezprozvanny;Mol. Pharmacol.,1995

5. Low threshold T-type calcium current in rat embryonic chromaffin cells;Bournaud;J. Physiol.,2001

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