Secreted protein acidic and rich in cysteine (SPARC) and a disintegrin and metalloproteinase with thrombospondin type 1 motif (ADAMTS1) increments by the renin-angiotensin system induce renal fibrosis in deoxycorticosterone acetate-salt hypertensive rats
Author:
Funder
Kyoto Pharmaceutical University
Hoansha Foundation
Kyoto University
Promotion and Mutual Aid Corporation for Private Schools of Japan
Publisher
Elsevier BV
Subject
Pharmacology
Reference45 articles.
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2. Protease profiling of liver fibrosis reveals the ADAM metallopeptidase with thrombospondin type 1 motif, 1 as a central activator of transforming growth factor beta;Bourd-Boittin;Hepatology,2011
3. The role of SPARC in extracellular matrix assembly;Bradshaw;J Cell Commun Signal,2009
4. SPARC-null mice display abnormalities in the dermis characterized by decreased collagen fibril diameter and reduced tensile strength;Bradshaw;J. Invest. Dermatol.,2003
5. SPARC-null mice exhibit accelerated cutaneous wound closure;Bradshaw;J. Histochem. Cytochem.,2002
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