Prostaglandins E1 and E2 inhibit lipopolysaccharide-induced interleukin-18 production in monocytes

Author:

Takahashi Hideo K.,Iwagaki Hiromi,Mori Shuji,Yoshino Tadashi,Tanaka Noriaki,Nishibori Masahiro

Publisher

Elsevier BV

Subject

Pharmacology

Reference19 articles.

1. Monocyte response to bacterial toxins, expression of cell surface receptors, and release of anti-inflammatory cytokines during sepsis;Astiz;J. Lab. Clin. Med.,1996

2. The origin and function of soluble CD14 in experimental bacterial meningitis;Cauwels;J. Immunol.,1999

3. International union of pharmacology classification of prostanoid receptors: properties, distribution and structure of the receptors and their subtypes;Coleman;Pharmacol. Rev.,1994

4. Endogenously opsonized particles divert prostanoid action from lethal to protective in models of experimental endotoxemia;Eierman;Proc. Natl. Acad. Sci. U. S. A.,1995

5. EP (E-prostanoid)2/EP4-receptor-dependent maturation of human monocyte-derived dendritic cells and induction of Th2 polarization;Kubo;J. Pharmacol. Exp. Ther.,2004

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